Summary |
· Fibrous pericardium stretches very slowly · Rapidly developing pericardial effusion compresses the heart · Reduced filling · Reduced output · General anaesthesia is often lethal, especially if IPPV |
Clinical signs |
· Beck’s triad: muffled sounds + distended neck veins + hypotension · Kussmaul sign: neck veins distend on inspiration (spont vent) · Pulsus paradoxus = audible heart beat + impalpable pulse in inspiration 1. Normal: ↓ITP -> pulmonary venous pooling -> ↓VR to LV 2. Abnormal: ↓ITP -> ↑VR to RV -> septal bulge -> ↓LV compliance |
ECG signs |
· ↑HR · Low voltage · Electrical alternans (↑↓QRS amplitude with oscillation in space) · ST-T changes |
Echo signs |
· Pericardium: o Effusion o ±Thickening · Cardiac chamber collapse o Systole before diastole o Right before left o Atria before ventricles · Inflow vessels: o Dilation o Diastolic flow reversal · Inspiratory changes: o Low output on inspiration (i.e. pulsus paradoxus) o Diastolic septal bounce |
Problems |
· Rate-dependent output · Pre-load dependent output · Intolerance of positive pressure ventilation · Intolerance of supine positioning · Associated disaster: trauma, dissection · Comorbidities: IHD, cardiomyopathy, cancer, inflammatory · Full stomach |
Goals |
· Prevent CVS collapse at induction o Percutaneous drainage prior o Minimise intrathoracic pressure o Full, fast, tight, sinus rhythm · Prevent rebound hypertension after drainage o Sympatholytics/vasodilators prepared · Prevent aspiration · Prevent awareness |
Approach |
· Assess for compromise o Toxic appearance o Large swing on A-line o Echo signs · If compromised: needle pericardiocentesis pre-induction o Must avoid pneumopericardium o Beware sedation -> upper airway obstruction -> big breath · If not compromised: careful induction of some sort · Consider having the patient prepped and draped |
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