Sepsis |
·
Life-threatening organ dysfunction
caused by
|
Septic shock |
· Sepsis · mAP <65 · Lactate >2 · Despite adequate volume resuscitation |
CVS |
· mAP · Pressor requirement |
Resp |
· P/F ratio |
CNS |
· GCS |
Kidney |
· Creatinine · Urine output |
Liver |
· Bilirubin |
Coag |
· Platelet |
(Quick Sofa: worry if any of a) RR >22 b) SBP <100 c) GCS <15)
Inflammation |
·
Not as simple as pro-inflammatory – i.e.
glucocorticoids aren’t curative · Early severe inflammation can kill · Late immune suppression can kill · Genomics may reveal different response phenotypes |
Coagulation |
· Coagulants upregulated · Anticoagulants consumed and downregulated · Microvascular thrombosis contributes to multi-organ dysfunction · Consumption may lead to DIC |
Endothelial injury |
· Vasodilation · Disrupted glycocalyx -> exudate (e.g. ARDS) |
Microcirculatory dysfunction |
· Heterogenous dysregulated expression of iNOS o A ‘steal’ phenomenon o Hyperperfusion if iNOS excess o Hypoperfusion of iNOS-deficient · Might be the reason for AKI |
Main things |
· Antimicrobial ASAP · Blood cultures beforehand |
Fluids |
· EGDT doesn’t help · Crystalloid is good enough for most · Albumin might be better in severe sepsis · Starches cause renal failure and death · Hb 70 is sufficient (TRISS) |
Vasoactives |
Vasopressors: · 1st line: noradrenaline · 2nd line: vasopressin (better for kidneys than ↑↑norad) Inotropes: · Dobutamine is safest Other: · Adrenaline is as good as noradrenaline but ↑HR, ↑lactate, ↑bsl · Dopamine causes arryhthmias · Role of inodilators is unclear |
Immunotherapy |
Glucocorticoids: · Reduce pressor requirement · Improves outcomes · Multiple adverse effects Other: · Trials ++ |
Anticoagulation |
· Safe · Makes no overall difference |
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