· Intro
· Problems
· Bleeding
· HITT
· Other (short term and long term)
Narrow therapeutic index |
· Need monitoring – aPTT · Aim 1.5-2.5x baseline approx |
Variable effect |
· Variable plasma protein concentration · Zero order kinetics at high dose · Variable elimination rate by metabolism (heparinase) > excretion (urine unchanged) · Heparin resistance o Variation in concentration of heparin-binding proteins (e.g. albumin) o ↑Heparin clearance (e.g. splenomegaly in liver disease) o ↓AT3 (e.g. DIC, bypass circuit, nephrosis, congenital) – Rx AT3 or FFP |
Risk |
· 1-5% if therapeutic anticoagulation · e.g. peptic ulcer bleed, haemorrhagic stroke |
Mechanism |
· Heparin binds and potentiates AT3 · AT3 inactivates IIa, Xa, IXa, XIa, VIIa-TF |
Risk |
· 1 in 200 therapeutic doses · 1 in 10,000 prophylactic doses · UFH >> LMWH |
Type 1 |
· 95-99% · ↓Platelet count by <10% · Onset <4 days · Non immune-mediate · Low risk complications |
Type 2 |
· 1-5% (<1% LMWH)) · Onset 4-14 days · ↓Platelet count by >50% or to <150 · Immune-mediated; IgG binds heparin + PF4 = hapten · Platelet activation and aggregation, thrombosis · 50% risk major thrombotic event: ATE, VTE · Risk major bleeding: haemorrhagic stroke, classic bilateral adrenal haemorrhage · Death 10-30% · Diagnosis: “4Ts”, heparin/PF4 assay · Rx: stop offender; start alternative e.g. lepirudin, argatroban · Do NOT start warfarin; risk skin necrosis, limb gangrene |
Short term |
· Hypotension (if rapid injection) – vasodilatation, venodilatation · Hypersensitivity · Hepatotoxicity · Suppression of aldosterone synthesis, ↑K+ · Skin necrosis |
Long term |
· Osteoporosis · Alopecia |
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