· Intro
· Angiotensinogen
· Renin
· Angiotensin 2
· Other angiotensins
· Aldosterone
Pathway |
· Note negative feedback o Direct: AT2 binds AGTR1 on granular cell -> ↑Ca2+ -> ↓renin release o Physiological: AT2 and aldosterone inhibit release of renin |
Overall function |
· Essential for fluid and electrolyte homeostasis and response to haemorrhage · Angiotensin 2: ↑ECF volume, ↑blood pressure · Aldosterone: ↑ECF [Na+] and osmolality, ↑ECF:ICF ratio |
Source |
· Large protein · Synthesised by the liver |
Function |
· Relatively inert circulating precursor for angiotensin 1 |
Source |
· Juxtaglomerular cell of the afferent arteriole |
Release stimuli |
· ↓Afferent arteriolar stretch · β1 agonism at JG cell · Low NaCl load to macula densa -> ↓adenosine + ↑PGE2 |
Kinetics |
· Released into circulation almost immediately upon stimulus · Duration 30-60 mins · Note it is the rate limiting factor due to constitutive presence of circulating angiotensinogen and pulmonary ACE |
Function |
· Enzymatic cleavage of antiotensinogen · Not itself vasoactive |
Kinetics |
· Produced within minutes of stimulus · Duration 1-2 mins (but more produced if renin present) · Metabolised by peptidases |
Cellular effect |
· Peptide hormone, 8 amino acids (angiotensin 1 is 10 amino acids) · Binds AGTR (Gq protein coupled receptor) · ↑IP3-> ↑ICF Ca2+; ↑DAG |
Vascular effect |
· Constriction of efferent > afferent arteriole o ↑GFP -> ↑GFR o ↓Peritubular capillary hydrostatic pressure -> ↑reabsorption · Generalised vasoconstriction >> venoconstriction, ↑SVR, ↑mAP |
Renal tubular effect |
· ↑Na+/H+ exchange in proximal tubule -> ↑Na+/H2O/HCO3- reabsorption -> ↑ECF volume and pH |
CNS effect |
· Hypothalamus: ↑ADH release · Circumventricular organs: thirst -> ↑H2O and NaCl consumption |
AT3 |
· Same as AT2 on aldosterone secretion · Same as AT2 at glomerulus · 60% of AT2’s systemic vasoconstriction |
AT1, AT4 |
· Also less active than AT2 |
Source |
· Zona glomerulosa of adrenal cortex · Derivative of cholesterol |
Release stimuli |
· CRH -> ACTH -> aldosterone (necessary for synthesis, minor effect on release) · Angiotensin 2 (major) · Hyperkalaemia (minor) |
Cellular effect |
· Steroid hormone · Binds cytosolic mineralocorticoid receptor, a nuclear transcription factor · Increases or decreases gene expression |
Renal tubular effect |
· Location: connecting tubule, collecting duct · Principal cell: o ↑Na+K+ATPase synthesis and activity o ↑Apical ANaC and ROMK activity · Type 1 intercalated cell: o ↑H+ATPase activity · Direct effects: o ↑ECF [Na+] and tonicity o ↑ECF pH o ↓ECF [K+] · Indirect effects: o ↑ECF:ICF volume ratio (due to osmosis) o ↑ADH release (due to hypertonicity) |
Colon and sweat glands |
· ↑Na+K+ATPase synthesis and activity -> ↑ENaC activity · ↑ECF [Na+] and tonicity |