· Production
· Transfer
· Carriage
· Variable CO2 affinity
Site |
· Mitochondrion |
Means |
· Krebs cycle, aerobic respiration · From carbohydrate, fat, amino acid metabolites |
Rate |
· ~200mL/min · Variable respiratory quotient (VCO2:VO2) o Average: 0.8 o Carbohydrate: 1.0 o Fat: 0.8 o Protein: 0.7 o Ketones: 0.7 · ↑VCO2: exercise, sepsis · ↓VCO2: hypothermia |
Fick’s law |
· Hence factors increasing CO2 transfer: ↑P1, ↓P2, ↑area, ↓thickness |
↑P1 |
· ↑metabolic rate -> ↑VCO2 |
↓P2 |
· ↑[Hb] -> ↑capacity for uptake · ↑volume of capillary blood -> ↑capacity for uptake · ↑cardiac output -> ↑rate of removal |
↑Area |
· ↑Capillarisation (e.g. adaptation to aerobic exercise) · ↑Recruitment and distension (e.g. exercise) |
↓Thickness |
· ↑Capillarisation -> ↓diffusion distance |
Total |
· CvO2 52mL/100mL venous blood · PvCO2 46mmHg |
As HCO3- (62%) |
· Location: o Haemoglobin (140g/L, 39 imidazoles per molecule) o Plasma proteins (70g/L, 13 imidazoles per molecule) · Purpose: o Maintain partial pressure gradient for CO2 from tissues into capillary blood · Mechanism: o CO2 + H2O < -> H+ + HCO3- (carbonic anhydrase in RBC) o KHb + H+ <-> HHb + K+ (buffering by imidazole groups of histidine residues) o HCO3- exchanged for Cl- at membrane (Hamburger effect, facilitates above reactions) |
As carbamino compound (28%) |
· Location: same · Purpose: same · Mechanism: NH2 + CO2 <-> NHCOO- + H+ |
As dissolved CO2 (10% |
· Amount dissolved = PaCO2 x k (Henry’s law) · Solubility coefficient k: 0.03mmol/L/mmHg at 37°C · Note ↑temp -> ↓solubility -> ↑PaCO2:CaCO2 |
Illustration |
|
Haldane effect |
· Higher uptake of CO2 and H+ in HHb cf. HbO2: o 70% of increment due to 3.5x ↑carbamino formation o 30% of increment due to ↑buffering (imidazole pKa 6.6 -> 8.2) |
Isohydric buffering |
· For each mol O2 unloaded by Hb, 0.7mol H+ can be added without ↓ plasma pH · Due to Haldane effect |
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